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Immunofluorescence staining of brain tissue examined the expression of tumor necrotic factor-alpha (TNF-, pro-inflammatory cytokine), glial fibrillary acidic protein (GFAP, a marker of activated astrocytes), sequestosome 1 (p62, marker of autophagic activity), voltage-dependent anion channel 1 (VDAC1, marker of mitochondrial stress), and NADPH Oxidase 4 (NOX4, marker of mitochondrial stress)

In experimental wound healing models, TB-500 treatment increases capillary density by 40-50% while improving functional blood flow restoration, demonstrating translation of cellular angiogenic activity into meaningful tissue-level perfusion benefits

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Conclusions Drug-induced liver injury was found to be a rare adverse effect of antibiotics in a population-based setting