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5.3.3 Improvement of myocardial metabolism, associated protective effects, and indirect impact on CAD GLP-1RAs enhance mitochondrial function in cardiomyocytes, mitigate oxidative stress and cellular damage, improve myocardial fatty acid metabolism, stabilize blood glucose levels, and regulate myocardial mitochondrial function via the reninangiotensinaldosterone system (156, 157)
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But so far, there has been comparatively less research on what happens when users stop taking the drugs
doi: 10.1016/j.asjsur.2022.11.112
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