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In summary, mitochondria-lysosome interactions play a critical role in regulating ROS and metabolic signaling, highlighting their importance not only in material turnover but also as key hubs for cellular stress sensing and the maintenance of functional homeostasis (Fig

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In states of insulin resistance, hepatic steatosis, and renal impairment, mitochondrial electron transport chain dysfunction drives the excessive production of ROS, which results in redox imbalance and metabolic stress (96, 97)

The activity of the natural killer cell-dependent and iron-dependent perforingranzyme pathway is blocked by iron overload, thereby decreasing the killing function of these cells

The protective role of EP300 in monocrotaline-induced pulmonary hypertension