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Based on the results obtained in the wet lab and in silico, they hypothesized that VIT-E inhibits LPx by reducing ROS and by increasing enzymatic antioxidant defense through binding of KEAP1, thus interfering with the NRF2-KEAP1 proteinprotein interaction and leading to ARE activation

discovered that adults with PWS had lower levels of vitamin D compared to the control group, regardless of differences in body fat (90)

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