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The rates of toxicities amongst patients who resumed single-agent anti-PD-1 were notably lower (for example, G3/4 colitis declined to 3%, and G3/4 hepatitis declined to 7%) [38]

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It has been demonstrated that in animals with ALDH4A1 mutation, vitamin E and vitamin C supplementation can reverse the inhibition of ATPase activity by hyperprolinemia, and appropriate use of antioxidants can also be helpful to reduce neurotoxicity [19]