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As newly identified metal ion-dependent programmed cell death modalities, ferroptosis and cuproptosis play critical roles in tumor metabolism, immune microenvironment remodeling, and therapeutic resistance, representing emerging research foci in OSCC

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They work by blocking the action of enzyme dipeptidyl peptidase 4 (DPP-4) in order to prevent the rapid degradation of endogenously released GLP-1, thus favoring glucose regulation