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Conversely, inflammatory conditions lead to an increase in gut expression of TRPV1 and TRPA1 [208]

Bhatia BK, Millsop JW, Debbaneh M, et al

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Experimental results demonstrated that apelin-13, an endogenous ligand of the G protein-coupled receptor angiotensin II protein J (APJ), increased DRP1 expression, inhibited fusion-related protein expression, and stimulated VSMC mitophagy and abnormal proliferation, which exacerbated the development of atherosclerosis [227]

Although these species are generated during normal cellular metabolism and participate in essential physiological processes like signal transduction and gene expression at low concentrations (Krishnamurthy et al., 2024), their excessive accumulation or insufficient scavenging leads to oxidative stress, causing damage to vital biomolecules and cellular structures