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The rationale is mechanistic: the two compounds operate through distinct but non-overlapping pathways, with tissue targets that complement rather than duplicate each other

Additionally, in the stroke-studies given after reperfusion initiation, after carotid arteries clamping, BPC 157 abrogated hippocampal ischemia/reperfusion injuries in rats [30]

In the end, the authors concluded that the high doses were able to raise the levels of GSH in blood neutrophils and the neutrophil count in the airways, with a significant reduction in elastase activity, the results being justified by the role of NAC in inhibiting the NF-B pathway, causing a reduction in IL-8

This keeps the work aligned with regulatory guidance while still allowing for detailed exploration of how the peptide behaves in various models